Journey

A.) I examined every patient and took down all their details, to try and understand how to treat. They all had the initial flu-like illness, and it didn’t seem much different from an average, viral flu (with some minor differences like the loss of smell/taste). In all the covid patients that I saw, I noticed by the 5th or 6th day they showed signs of recovery. They got their appetites back, their fever broke, and they started to feel better – but there was a sub-set of patients – who would present breathlessness – who came back to me, and there were a few very unusual things. The first thing I noticed was that every patient that developed breathlessness in the first wave, developed it exactly a week after their first symptom. So they would go through the standard flu-like illness, start feeling better, start going back to normal, but exactly one week later, they would get up feeling fatigued, breathless, and by the evening were showing signs of saturation-dropping. So it was very strange that on the 8th day, you were having patients with breathlessness, who on the day before, were perfectly fine. So I knew I was dealing with something that had 2 completely different pathogenesis.

The sub-set of patients with curious day 8 worsening:

So I started looking at the 2nd part. Why was there a sub-set of patients who came back on the 8th day, breathless? Looking at the group, it didn’t seem like they had a predisposition to comorbidities. Yes, they were slightly older – but I’ve had older patients that recovered with severe comorbidities that still recovered relatively quickly. I’ve had patients in their 50’s, absolutely healthy, no comorbidities, that presented on the 8th day with very severe illness. So I couldn’t see a pattern really to comorbidity.

Examining them was unusual:
A typical viral pneumonia is a progressive-illness. It starts with an upper-respiratory infection, spreads into a bronchitis, progresses into a pneumonia, and that’s where the breathlessness sets in. So typically, you would see the progression on a daily-basis to pneumonia. However with this sub-set of patients, they were perfectly fine the day before, so there wasn’t that typical progression that you would see with a viral pneumonia.

The second thing to note, is that in typical viral pneumonia, patients are acutely ill. Whereas with this sub-set, they were breathless & fatigued, but they weren’t acutely ill. If you took a step-back from afar, you would think that patient is perfectly fine. So it wasn’t the typical presentation of a pneumonia.

The third thing I noticed in clinical examination, these patients had no restriction to air-flow. So air was entering and leaving their lungs unrestricted. Unlike a pneumonia where there is a restriction. This sub-set just couldn’t take a “deep breathe”. The breathing was rapid and shallow – they were panting.

Some patients on the 8th day had mild symptoms and others had more moderate symptoms, and the only thing that made sense from a pathology perspective was an allergic reaction to an allergen, triggered on the 8th day. So for those patients, I gave steroids, and they recovered within 2-3 days.

So understanding that it was some kind of hyper-sensitivity, I decided to treat as such. So the next patient I also added an anti-histamine, and the very next day she was perfectly fine. But I only gave her 1 day’s worth of medication, and when you treat an allergic-reaction, it’s got to be suppressed until your muscles actually calm down – if you withdraw treatment too quickly, you’ll have a rebound. So I asked my staff to inform her if the breathlessness comes back, she needs to report back immediately, and the following day, the breathlessness resurfaced, and she was started on a full course of antihistamine and she promptly recovered.

From that patient, I was under the impression, that we’re not dealing with the covid pneumonia, we are rather dealing with an allergen- instigated: Hypersensitivity Pneumonitis.

On high-definition CT and X-Ray (which is the standard of looking at pneumonia), these 2 conditions cannot be told apart. So I’ve been under the impression from the start of this pandemic, that the globe has misdiagnosed this illness, as a covid-pneumonia.

Looking at the mediators released when you have an allergic reaction, the 4 main mediators that get released are: histamines, leukotrienes, prostaglandins, and platelet-activating factor.

My protocol of treatment followed that theory early-on.

To the antihistamines, I added Montelukast to deal with the leukotrienes, and added an Anticoagulant (Aspirin as a blood-thinner) for the platelet-activating factor.

If you are treating pneumonia, you have to be very cautious with the steroids, however if you are treating a Hypersensitivity Pneumonitis, the high-dose of the steriod is vitally important. You have to turn the reaction around before it spirals out of control.

It is dependant on your genetic pre-disposition to allergy, rather than any co-morbidity.

Those who had mild symptoms on the 8th day (like a bee-sting), they had mild transient irration in their lung, and that would resolve spontaneously.

The second sub-set would be those that had moderate-reactions, (like a bee-sting that causes a rash throughout your body), and if you left that to be settled on its own, it might take a very long while for your body to rectify that, and that is what we saw as Long-Covid. Long-covid in my opinion is just a moderate allergic reaction triggered on the 8th day, that has been inappropriately treated and left unchecked for too long, so it spirals out of control.

The third sub-set are those that are severely-allergic to this allergen, and would have a severe-reaction. That would spiral very quickly and would result in severe inflammation in your lungs, and if unchecked would result in a Cytokine storm where they’d put you on a ventilator.

The dose of steroid in the first wave had to be titrated up, and I came to an average dose of about 80mg of Prednisolone as the minimum effective dose. Anything below that, didn’t seem to work.

In the first wave, I had no deaths or hospitalizations or oxygen, because everyone, no matter how severe, seemed to recover by the next day, and I had not a single patient develop long-covid or any complications.

In the second wave, I was now examining long-covid patients from the first-wave (not treated with my modality), I took the opportunity to record bio-markers to prove my theory of Type I Hypersensitivity.

I needed to show on the 8th day the release of Histamine or Tryptase or an elevated level of Immunoglobulin E (the Immunoglobulin involved in allergic reactions) as these are the common markers, however Tryptase was an expensive test to do, Histamine required a 24 hr urine sample to be collected – and would have to defer treatment which would endanger them, so I couldn’t do those tests to prove the 8th day.

However with the long-covid patients, I knew they were exposed to the allergen long enough to have been given sufficient time to have elevated IgG Levels. So I tested them and every single one of them had elevated IgG levels to the point where the normal values ranged up to 100 – whereas the Long-Covid levels were values around 5,000, and I used that as a marker of severity and of course a marker for recovery.

So I put them on a protocol and all of them within 3-4 days, irrespective of how long they were suffering long-covid, showed signs of good recovery. However when you treat of long-covid, because of the length of the injury, some things are irrepairable.

When we got into the second wave, the South African variant, the 8th day remained exactly the same, but I was starting to see new gastro-symptoms, runny-tummy’s, cramping, nausea, vomiting, however on the 8th day, it was more re-emergence of diarrhoea and an overwhelming sense of fatigue (when the previous day they were perfectly fine). The allergen triggering this process with the Spike Protein. It was the only thing that changed on this variant, which caused those 3 symptoms to change.

The Spike Protein is what gives a virus, its affinity for a particular host.
We’ve been exposed to coronaviruses throughout history but this is the first time I’ve seen an “allergic” type of presentation. So usually when you are exposed to new environments, there’s a risk that you will be allergic to something, seeing that it is a new-or-unusual exposure.

I added Plasmoquine – a sub-set of HCQ, just as an antiviral, and it worked well and all patients recovered within 2 or 3 days. That was reserved for a few patients that seemed to have high viral loads.

The rest were treated symptomatically on what might transpire on the 8th day.

So the treatment modality of the 4 waves we’ve had, has not changed. Every patient is taught about the 8th day.

With an allergic reaction, quick, aggressive, timely intervention is the most vital thing.

In the third wave, the Delta variant, we saw exactly the same thing. There were slight changes in the initial presentation. Changes in the severity of the Hypersensitivity allergy triggered on the 8th day, and changes in the symptoms, but the timeline remained consistent irrespective of which variant I was dealing with.

Viruses tend to have a very set pattern when they infect a human, unlike bacteria. Bacteria can grow indefinitely. Viruses run through so many cycles over a period of so many days, and then they eventually are self-limited by an immune response.

All the mortality and severity of this virus, lies in the 2nd part.

Nobody dies of the first 7 days of viral exposure. It’s those that have a severe allergic reaction post 8 day, that have the highest risk of mortality. Those are the ones that would end up in hospital.

Why were over 55’s more prone? Over 55 year old patients had been previously exposed to something very similar to this coronavirus. To put that in perspective, if you are allergic to a bee-sting, the first time you will not have a clinical reaction to it, because you don’t have the antibodies that would trigger the allergic reaction. But on that first exposure, you could use a specific sub-type of IgG E for that particular allergen. The 2nd time, you are exposed to it, you now have the IgG to mount an inappropriate allergic response to it.

Younger people weren’t reacting during the first wave, simply because they have never been exposed to it – it would be their first exposure to the allergen. However, during that first exposure, those that were allergic, would likely be sensitized and would have an allergic reaction with subsequent exposure.

Dr. Shankara Chetty

In the third wave, I was exposed to vaccination patients. If you understand the underlying pathology, then you can postulate what might be the case. What I’m seeing in vaccinated patients, with the Pfizer / mRNA vaccine, I noticed that once the vaccination programme had started, I was seeing patients presenting to me, a large sub-set 7-10 days after injection, came the onset of illness with either sore throat or the onset of breathlessness, and so there were too many of these patients presenting after vaccination, testing positive for Covid for it to be ‘just a coincidence’. In that sub-set of those patients, there were some who on their 2nd, 3rd, or 4th day of illness, suddenly deteriorated and their oxygen dropped – and that has never happened with the unvaccinated patients – irrespective of the variant.

I felt I was dealing with a “spike protein illness” – an allergic illness dealing with the spike protein.

I classified it early on as “spike protein illness”. The vaccinated had missed the viral-phase of the illness completely, the vaccine had forced their bodies to produce the spike proteins, and when you’ve reached an appropriate concentration, it triggered a reaction.

So these patients on the first day of illness, were actually already onto the day 8 Hypersensitivity Pneumonitis phase of the illness.

They had missed the viral phase completely and likely to be shedding the spike protein or messenger RNA for the PCR test to be turning positive.

With the unvaccinated patient, on the 8th day I do a few biomarkers – Interleukin 6, D-dimer, CRP. Which gives me a good indication of the 2nd phase. They tend to be slightly elevated in the first phase of the illness, but from the 8th day, take off exponentially. So if I see those elevated markers, I’m 100% sure that this patient has gone into the Hypersensitivity phase, and I use those markers to gauge the recovery. The aim was to get them back to baseline as quickly as possible.

Now with these patients that presented after vaccination, I decided to do the very same biomarkers but do it on the day they present with illness, and I saw the elevations, which clarified for me that they were already into the Hypersensitivity phase, and had missed the viral phase completely.

So those patients that were crashing 2 or 3 days into the illness, that were deteriorating with Hypoxia and the rest, were those that were the most allergic to the spike protein, and of course, the vaccine takes a little while to build the spike protein in the body, so it looked like there were a 7-10 day odd ‘lag’ between the injection and the reaction, and that ‘lag’ would allow IgE to develop.

So with those patients that presented early-on with vaccine-reactions, I did IgE levels on them, and every single one of them who had an allergic reaction, had drastically elevated IgE levels. So their IgE levels became the marker as to the severity of the reaction that might occur. So those that have high elevated IgE levels needed to be watched very closely – they were the ones that could deteriorate very rapidly. The treatment of those patients a week after vaccination, was exactly the same as I use from the 8th day on the unvaccinated – steroids, antihistamines, Montelukast, anticoagulant – and all of them showed signs of recovery.

So when I look at the vaccines itself, we’re dealing with a few different mechanisms:
The immunity is different is from the allergy. So the vaccines itself are meant to stimulate an immune-response, which is supposed to give you immunity against infection and transmission, however the vaccine as well exposes you to the spike protein, which is an allergen. So the vaccine has the ability to develop a measure of tolerance to an allergen. So that would be where the vaccine gives us the benefit of improvements in severe illness and death compared to any immune-mediated resistance to a virus itself.

I’ve seen breakthrough infections constantly from what we suspect is Covid-illness but clearly is Spike Protein illness, and then I’ve had a sub-set of patients that presented a month or two after vaccination with Covid. Now those patients are truly having breakthrough infections. We’ve given them sufficient time after vaccination to develop a robust immune-response to the virus, and if they develop infections subsequently, then that’s a ‘breakthrough infection’. And in those patients that present almost a month after vaccination, they have the typical evolution of the illness. They have the first 7 days of viral-illness, and then on the 8th day show signs of worsening – the same that we see in unvaccinated patients.

So I’m of the opinion that the vaccine itself, provide us with no “immune” benefit. Just today alone I’ve seen 8 patients that are fully-vaccinated that presented with Covid. So clearly the vaccine does not stop infection or transmission. So as a vaccine, it has failed – it does not do its job. But where we see the improvements in the clinical outcome, now there’s this claim that vaccines prevent severe illness or death – that is due to the vaccine being able to stimulate a measure of tolerance to an allergen or desensitization.

So when a patient is exposed to the Spike Protein – if they are allergic – that low-dose of spike protein that they’re exposed to will give them a measure of tolerance, so that when they are exposed to the allergen through a viral infection, they do not develop the same severity of Hypersensitivity reaction, and that is where the benefit is – in the decrease of severity of symptoms due to desensitization to an allergen – not to any immune-mediated benefit.

And that desensitization will only persist for as long as there is exposure to the allergen. So we’ve seen 3 or 4 months post-vaccination that vaccinated people start to have severe illness again, that’s a good indication that they’re no longer exposed to the spike protein – so the desensitization has stopped, the tolerance has waned, so upon exposure, they will have a severe reaction again.

The vaccine doesn’t have the spike protein – it’s a messenger RNA that gets your body to make the spike protein, so the exposure to the spike protein post-vaccination will be delayed.

If someone has an immediate reaction to the vaccine, the immediate reaction can occur to messenger RNA, Polyethylene glycol, graphene oxide, and all the other adjuvants and additives in the vaccine itself. Hypersensitivity can occur with a wide diversity of ingredients. However, the allergy to the Spike Protein would be restricted to those patients who are allergic and would be reasonably dose-dependent.

When you look at the myocarditis and all those kinds of immediate side-effects, those might not be related to hypersensitivity but more related to the biologic effect of the Spike Protein itself. This has been a push from the start, because I wanted research to stop focusing on the virus and start focusing on the Spike Protein, so we could understand its biological effect on the body.

There’s a lot of information that’s come out since about its biological effect:
We’ve seen the endothelial injuries, the effect of the myocardium in the heart, and other pathologies like prions, so we understand there will be neurological illness because of that and a host of increases in Alzheimer’s and Dementia. We’ve also seen similarities to other pathogenic proteins related to HIV, and so we expect this to have an immunosuppressive effect, subsequently causing the emergence of latent infections and the re-emergence of cancers that were in remission. We’ve also seen that the spike protein has the ability to enter the cell nucleus, and with its inhibition of the BRCA enzyme, which can actually prevent DNA-repair, that has some grave long-term consequences in the inability of cells to repair, and those that survive damage would likely force cancerous growths.

The spike protein is a membrane protein, and so it should be expressed on the cell that produces it; that cell will be recognized as foreign to the rest of the body, and that would trigger a host of autoimmune responses depending on the tissue that’s actually making the spike protein and depending on where it’s actually been distributed.

The spike protein has a wide diversity of toxic biologic effects on the body, and any one of those effects could come into play at any point in time. So, yes, there are immediate responses, but if it’s an immediate response, it’s very likely to be a response to one of the adjuvants or the messenger RNA, because I’ve seen this allergic proclivity show up 7-10 days after vaccination, and I believe it takes 7-10 days for a sufficient amount of the spike protein to be built to trigger this allergic process.

Dr. Shankara Chetty

If you look at another chemical, like Penicillin. Penicillin acts like an antibiotic. To get that biologic effect from Penicillin, you need to have a full course—a certain length of exposure—to have that biologic effect. Now, if I, like the virus, gave everyone on the planet a single dose of Penicillin and then isolated them for 14 days, I would not see the biologic effect—it would not act as an antibiotic—because it was a single dose. Those who are allergic to Penicillin, on the other hand, would perish if they were not treated.

And I think that’s what’s happened with the virus. People who were allergic, without understanding the illness, and because of the delay in early-treatment and intervention, have died.

When you take a vaccine, you are exposed to the spike protein for a longer period of time and possibly at a higher dose, and thus you would see its biologic effects, similar to a full course of Penicillin, and thus there is a need to understand the spike protein’s effects on the human body.

If we look at the VAERS system and Long-Covid – those would be the two-reporting where patients were exposed to the Spike Protein – we might not see the numbers / the absolute values because of the under-reporting and non-reporting of symptoms and deaths, but we can look at the spread of pathology that we see. And if you look at all the reports on the VAERS system, the prediction of what the Spike Protein might do as a biologically-active substance is exactly the spread of pathology that we see in the VAERS system: Endothelial injuries, which cause strokes, heart attacks, blood clots, myocarditis, neuropathies, increases in Alzheimer’s and dementia, immunosuppression, the emergence of latent viruses, the re-emergence of cancers, the spiraling of auto-immune conditions, and Guillain-Barré syndrome – previously uncommon conditions – have now become “the norm.” The spike protein is very likely the culprit here.

All-cause mortality is vitally important to look at, and of course excess deaths.

If we don’t know what’s in the vaccine, then every single side-effect is considered a vaccine side effect until we prove otherwise—not the other way around. So no cherry-picking side-effects—we’ll never figure that out.

Dr. Shankara Chetty

Individuals who had a severe reaction to COVID are more likely to have a severe reaction to the vaccine. The first dose of vaccine produces a mild reaction, while the second dose produces a much more severe reaction. However, I’ve had patients who have had severe reactions to natural infections but had no reaction to the vaccine, and I find that very confounding and confusing. We’re also aware that there are inconsistencies in the vaccines. The doses in the vials are not the same. Some have just normal saline. So I think that’s more likely to be due to the inconsistencies in the vaccines than any inconsistencies in the response to the illness.

The first dose is a mild dose, and you’re not sure what patients are actually getting in the first dose. In all patients, the second dose seems to cause far more severe reactions. I’ve had many patients die from the vaccines. I’ve seen a lot of neuropathies, a lot of strokes, and a lot of cardiac issues. Unfortunately, the medical fraternity refuses to accept that. I’ve had patients who were told that it was “not the vaccine,” even though the side effects occurred a day or two after the vaccine. So I think that the information that we collect from this is never going to be good data because we’re not looking at “every case,” we’re not looking to understand “every case,” and we’re just trying to “justify the vaccine” by brushing a lot under the carpet.

/// Q.) I’ve heard that Hepatitis B is similar to the coronavirus? That half of the spike protein is similar. Is there a difference for those already vaccinated against HepB?

A.) The problem is with the spiked protein and its ability to suppress immunity. There are a lot of latent viruses in our body that are suppressed by a good immune response, and that immune-response tends to be lifelong. However, if you suppress that immune response, these latent-viruses are going to show up.

India is having an outbreak of dengue at the moment—that is very strange. Strange after a mass vaccination campaign.

Dr. Shankara Chetty

At the start of the pandemic, I thought that messenger RNA was the stupidest technology to use to make this vaccine.

We’re taking messenger RNA, putting it in a cell, and that cell has to make the spike protein, which is coded by that mRNA, that spike has to be recognized as “foreign” and trigger an immune response. That immune response is meant to build specific antibodies to that protein, and if those antibodies are neutralizing and you are exposed to a variant that has the same type of spike protein, then those neutralizing antibodies will kill the virus, and that is the way in which the mRNA is “meant” to work.

So I asked a question.
Messenger RNA was meant to be stored at minus 90 degrees Celsius.
We’re not sure of its biodistribution in the body.
We’re not sure how long-lived it’s going to be.
We’re not sure of how much spike protein it will make.
We’re not sure how long it will make spike proteins.

So we have all these variables that make the vaccine difficult to monitor. But the end product, is a neutralizing antibody to the spike protein. So why didn’t we inject people with the spike protein directly?

We would’ve got to the same end-point.

The spike protein would’ve triggered an immediate response, which would’ve caused the development of antibodies to the spike protein, which would’ve given us the same endpoint without the mRNA involved, and nobody could answer that.

It would be easier to manufacture.
It wouldn’t require a specific minus 90-degree Pfizer-fridge.
We could dose it perfectly based on patients body-weight.
We would know it’s half-life and exactly how long it would be eliminated from the body.

It’s something we could control far more effectively.

So what was the need to use Messenger RNA to make the Spike Proteins?

The best vaccine would be a live-attenuated vaccine.
So take the mildest variant of coronavirus, and use it as a candidate to make a live-attenuated vaccine.

That vaccine would cause a mild illness, and you would develop immunity once you recovered.
Seeing that it caused a mild illness, it would be self-spreading.

So you won’t be required to vaccinate the entire population; whoever you vaccinate, just give them advice that when they develop symptoms, they must go play in a crowd, and they’ll spread that vaccine to every person around them.

So we create an artificial mild-pandemic, that would spread very quickly, and we’ll develop natural immunity without the mortality.

With Omnicom, God seems to have given us that. We have a mild variant that’s highly contagious without the mortality, and Omicron is better than the vaccine in many ways, so it should be the replacement for mass-vaccination. We should all get Omicron and get natural immunity.

Closing our borders and trying to vaccinate the population is the most nonsensical thing to do – it’s stupid – open your borders and get herd immunity without lifting a finger.

Dr. Shankara Chetty

Whether it’s mRNA or nanotechnology, it is absolutely, wholly, unnecessary. We are spending our time chasing and killing bees, and we’re not treating the people who are allergic to the ‘sting’. It’s a waste of our time.

If we concentrated on the mortality and morbidity like we should’ve done from the start with early-treatment to build understanding about its pathology that we see, it would make vaccines unnecessary.

If we focused on early treatment to reduce COVID mortality and morbidity, there would be no need to rush a half-baked vaccine to market on a global scale. Vaccines are completely the wrong-direction to take when you talk of “covid-illness”. I’m not an antivaxxer; I have all my vaccinations. This is inappropriate science, and I cannot justify anything that would injure my patient. It’s the first principle, “Do No Harm,” so as long as I see a vaccine’s ability to cause injury, I’ve got to weigh that risk up against the benefits it might provide to that patient, and quite frankly, with early treatment, no one is really at risk. If you do not treat, then everyone’s at risk.

When you look at the comorbidities as well, a bee doesn’t care if you are fat or thin or whether you have diabetes or high blood pressure; all it cares is that you are allergic to its sting. The comorbidities do play a part, but they play a part in the recovery of the illness, not in the pathology of the illness itself. So if you are a diabetic and you get a bee sting, you have a higher risk of severe illness, but the bee sting is not going to kill you because you are “diabetic,” it will kill you because you are “allergic to a bee sting.”

If you look at the vaccines, they should stop infection and transmission, and that’s what gives a vaccine a “group benefit.” So if you take a vaccine and it stops transmission and infection, you actually protect me, and that’s the population-benefit of a vaccine, so it should stop transmission; we don’t talk about “decrease.” If they claim that it reduces transmission, that is questionable. If a person is highly infectious and lives across the street from me, it doesn’t affect me at all. If a person is mildly infectious and in the middle of the dance floor, he will probably infect everyone around him. Either it stops transmission and infection, or it doesn’t. Now clearly, Pfizer and the rest have acknowledged that it doesn’t stop transmission or infection, so quite frankly, that means that you haven’t proven a group benefit to your vaccination.

Saying that it prevents severe illness and death is a “therapeutic benefit.” My treatment prevents severe illness and death, but I don’t expose the entire planet to the side effects of my treatment. It’s limited to those who are unwell. I don’t go around giving people paracetamol in anticipation of a headache tomorrow, and then when that wears off, give them another just in case the headache hasn’t appeared yet; they’d get side effects from that; that’s where we’re going with these vaccinations. Repeated doses to try and prevent you from getting a severe reaction to the spike protein. So you haven’t yet proven a group-benefit.

The prevention of severe illness and death is also an individual benefit, so if you take the vaccine, you might not be severely ill or die when you get covid-illness, but that benefit is not transferable to me; it has no bearing on me whatsoever. So if I treat you for the flu, I don’t expect your neighbour to improve with your treatment, and I can’t understand, without this population-based benefit, the justification for mandating it globally. There is absolutely no logic to mandating something to a group where you haven’t shown a group benefit.

If the doctors know what to do and the people know what to look for, I don’t need anyone’s permission to save lives. I knew from the start that patient education is vitally important. It is how they’ve coerced the population—through fear and manipulation—so getting people to understand what’s going on is vitally important.

1hr 20min:
Great Vaccine Analogy: “Imagine skydiving with holes in your parachute and a few dead bodies on the ground, and you’re telling me, “Nothing’s wrong, don’t look there, just jump,” and I’ll give you a donut.”

Vaccine-hesitancy is warranted, you’ve got to be an idiot to do this.

Dr. Shankara Chetty